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The 42nd Congress of the International Society of Oncology and Biomarkers "ONCOLOGY IN THE BIOMARKER ERA: Biology-Diagnostics-Therapy" ISOBM 2015 : October 3-7, 2015, Hotel Belvedere, Zakopane, POLAND.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

[No authors listed]
PMID: 26373736
Tumour Biol. 2015 Sep;36:1-88. doi: 10.1007/s13277-015-4009-7.

No abstract available.

Cite
The 42nd Congress of the International Society of Oncology and Biomarkers "ONCOLOGY IN THE BIOMARKER ERA: Biology-Diagnostics-Therapy" ISOBM 2015 : October 3-7, 2015, Hotel Belvedere, Zakopane, POLAND. Tumour Biol. 2015;36:1-88doi: 10.1007/s13277-015-4009-7.
(2015). The 42nd Congress of the International Society of Oncology and Biomarkers "ONCOLOGY IN THE BIOMARKER ERA: Biology-Diagnostics-Therapy" ISOBM 2015 : October 3-7, 2015, Hotel Belvedere, Zakopane, POLAND. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, 361-88. https://doi.org/10.1007/s13277-015-4009-7
"The 42nd Congress of the International Society of Oncology and Biomarkers "ONCOLOGY IN THE BIOMARKER ERA: Biology-Diagnostics-Therapy" ISOBM 2015 : October 3-7, 2015, Hotel Belvedere, Zakopane, POLAND." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. 36 (2015): 1-88. doi: https://doi.org/10.1007/s13277-015-4009-7
The 42nd Congress of the International Society of Oncology and Biomarkers "ONCOLOGY IN THE BIOMARKER ERA: Biology-Diagnostics-Therapy" ISOBM 2015 : October 3-7, 2015, Hotel Belvedere, Zakopane, POLAND. Tumour Biol. 2015 Sep;36:1-88. doi: 10.1007/s13277-015-4009-7. PMID: 26373736.
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Overexpression of miR-100 inhibits cancer growth, migration, and chemosensitivity in human NSCLC cells through fibroblast growth factor receptor 3.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Luo J, Chen B, Ji XX, Zhou SW, Zheng D.
PMID: 26314855
Tumour Biol. 2015 Aug 28; doi: 10.1007/s13277-015-3850-z. Epub 2015 Aug 28.

Nonsmall cell lung cancer (NSCLC) is a commonly occurring lung cancer. A combination of molecular biological treatments with regular chemotherapy may result in improved therapeutic outcome. Here, we reported significantly higher levels of fibroblast growth factor receptor 3 (FGFR3)...

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Luo J, Chen B, Ji XX, et al. Overexpression of miR-100 inhibits cancer growth, migration, and chemosensitivity in human NSCLC cells through fibroblast growth factor receptor 3. Tumour Biol. 2015;doi: 10.1007/s13277-015-3850-z.
Luo, J., Chen, B., Ji, X. X., Zhou, S. W., & Zheng, D. (2015). Overexpression of miR-100 inhibits cancer growth, migration, and chemosensitivity in human NSCLC cells through fibroblast growth factor receptor 3. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, . https://doi.org/10.1007/s13277-015-3850-z
Luo, Jie, et al. "Overexpression of miR-100 inhibits cancer growth, migration, and chemosensitivity in human NSCLC cells through fibroblast growth factor receptor 3." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. (2015). doi: https://doi.org/10.1007/s13277-015-3850-z
Luo J, Chen B, Ji XX, Zhou SW, Zheng D. Overexpression of miR-100 inhibits cancer growth, migration, and chemosensitivity in human NSCLC cells through fibroblast growth factor receptor 3. Tumour Biol. 2015 Aug 28; doi: 10.1007/s13277-015-3850-z. Epub 2015 Aug 28. PMID: 26314855.
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Association of single-nucleotide polymorphisms of high-mobility group box 1 with susceptibility and clinicopathological characteristics of uterine cervical neoplasia in Taiwanese women.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Wu HH, Liu YF, Yang SF, Lin WL, Chen SC, Han CP, Wang HL, Lin LY, Wang PH.
PMID: 27704361
Tumour Biol. 2016 Oct 04; doi: 10.1007/s13277-016-5408-0. Epub 2016 Oct 04.

To date, no study associated the genetic polymorphisms of high-mobility group box 1 protein (HMGB1) with the development of uterine cervical cancer. We therefore conducted this study to investigate the associations of HMGB1 single-nucleotide polymorphisms (SNPs) with cervical carcinogenesis...

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Wu HH, Liu YF, Yang SF, et al. Association of single-nucleotide polymorphisms of high-mobility group box 1 with susceptibility and clinicopathological characteristics of uterine cervical neoplasia in Taiwanese women. Tumour Biol. 2016;doi: 10.1007/s13277-016-5408-0.
Wu, H. H., Liu, Y. F., Yang, S. F., Lin, W. L., Chen, S. C., Han, C. P., Wang, H. L., Lin, L. Y., & Wang, P. H. (2016). Association of single-nucleotide polymorphisms of high-mobility group box 1 with susceptibility and clinicopathological characteristics of uterine cervical neoplasia in Taiwanese women. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, . https://doi.org/10.1007/s13277-016-5408-0
Wu, Hsin-Hung, et al. "Association of single-nucleotide polymorphisms of high-mobility group box 1 with susceptibility and clinicopathological characteristics of uterine cervical neoplasia in Taiwanese women." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. (2016). doi: https://doi.org/10.1007/s13277-016-5408-0
Wu HH, Liu YF, Yang SF, Lin WL, Chen SC, Han CP, Wang HL, Lin LY, Wang PH. Association of single-nucleotide polymorphisms of high-mobility group box 1 with susceptibility and clinicopathological characteristics of uterine cervical neoplasia in Taiwanese women. Tumour Biol. 2016 Oct 04; doi: 10.1007/s13277-016-5408-0. Epub 2016 Oct 04. PMID: 27704361.
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MiR-153 regulates metastases of gastric cancer through Snail.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Wang Z, Liu C.
PMID: 26232914
Tumour Biol. 2015 Aug 02; doi: 10.1007/s13277-015-3846-8. Epub 2015 Aug 02.

MicroRNAs (miRNAs) play critical roles in tumorigenesis and cancer metastases of gastric carcinoma (GC). Recently, miR-153 was reported as a tumor suppressor miRNA silenced in GC, whereas the underlying mechanisms are much lacking. Here, we reported that miR-153 levels...

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Wang Z, Liu C. MiR-153 regulates metastases of gastric cancer through Snail. Tumour Biol. 2015;doi: 10.1007/s13277-015-3846-8.
Wang, Z., & Liu, C. (2015). MiR-153 regulates metastases of gastric cancer through Snail. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, . https://doi.org/10.1007/s13277-015-3846-8
Wang, Zhongli, and Liu, Chao. "MiR-153 regulates metastases of gastric cancer through Snail." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. (2015). doi: https://doi.org/10.1007/s13277-015-3846-8
Wang Z, Liu C. MiR-153 regulates metastases of gastric cancer through Snail. Tumour Biol. 2015 Aug 02; doi: 10.1007/s13277-015-3846-8. Epub 2015 Aug 02. PMID: 26232914.
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Downregulation of Smurf2 ubiquitin ligase in pancreatic cancer cells reversed TGF-β-induced tumor formation.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Wu B, Guo B, Kang J, Deng X, Fan Y, Zhang X, Ai K.
PMID: 27730540
Tumour Biol. 2016 Oct 11; doi: 10.1007/s13277-016-5432-0. Epub 2016 Oct 11.

Smad ubiquitin regulatory factor 2 (Smurf2) is an E3 ubiquitin ligase that regulates transforming growth factor β (TGF-β)/Smad signaling and is implicated in a wide range of cellular responses. However, the exact mechanism whereby Smurf2 controls TGF-β-induced signaling pathways...

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Wu B, Guo B, Kang J, et al. Downregulation of Smurf2 ubiquitin ligase in pancreatic cancer cells reversed TGF-β-induced tumor formation. Tumour Biol. 2016;doi: 10.1007/s13277-016-5432-0.
Wu, B., Guo, B., Kang, J., Deng, X., Fan, Y., Zhang, X., & Ai, K. (2016). Downregulation of Smurf2 ubiquitin ligase in pancreatic cancer cells reversed TGF-β-induced tumor formation. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, . https://doi.org/10.1007/s13277-016-5432-0
Wu, Bo, et al. "Downregulation of Smurf2 ubiquitin ligase in pancreatic cancer cells reversed TGF-β-induced tumor formation." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. (2016). doi: https://doi.org/10.1007/s13277-016-5432-0
Wu B, Guo B, Kang J, Deng X, Fan Y, Zhang X, Ai K. Downregulation of Smurf2 ubiquitin ligase in pancreatic cancer cells reversed TGF-β-induced tumor formation. Tumour Biol. 2016 Oct 11; doi: 10.1007/s13277-016-5432-0. Epub 2016 Oct 11. PMID: 27730540.
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MicroRNA-133a suppresses the proliferation, migration, and invasion of laryngeal carcinoma cells by targeting CD47.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Li H, Wang Y, Li YZ.
PMID: 27730543
Tumour Biol. 2016 Oct 11; doi: 10.1007/s13277-016-5451-x. Epub 2016 Oct 11.

The study aims to investigate the possible mechanisms of microRNA-133a (miR-133a) targeting CD47 on cell proliferation, apoptosis, migration, and invasion in laryngeal carcinoma. Forty-two laryngeal carcinoma tissue specimens confirmed by pathological examination from laryngeal carcinoma patients as the case...

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Li H, Wang Y, Li YZ. MicroRNA-133a suppresses the proliferation, migration, and invasion of laryngeal carcinoma cells by targeting CD47. Tumour Biol. 2016;doi: 10.1007/s13277-016-5451-x.
Li, H., Wang, Y., & Li, Y. Z. (2016). MicroRNA-133a suppresses the proliferation, migration, and invasion of laryngeal carcinoma cells by targeting CD47. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, . https://doi.org/10.1007/s13277-016-5451-x
Li, Hui, et al. "MicroRNA-133a suppresses the proliferation, migration, and invasion of laryngeal carcinoma cells by targeting CD47." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. (2016). doi: https://doi.org/10.1007/s13277-016-5451-x
Li H, Wang Y, Li YZ. MicroRNA-133a suppresses the proliferation, migration, and invasion of laryngeal carcinoma cells by targeting CD47. Tumour Biol. 2016 Oct 11; doi: 10.1007/s13277-016-5451-x. Epub 2016 Oct 11. PMID: 27730543.
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Erratum to: RNA interference-mediated knockdown of RhoGDI2 induces the migration and invasion of human lung cancer A549 cells via activating the PI3K/Akt pathway.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Niu H, Wu B, Peng Y, Jiang H, Zhang Y, Wang J, Zhang Y, He P.
PMID: 25487616
Tumour Biol. 2015 Jan;36(1):459. doi: 10.1007/s13277-014-2904-y.

No abstract available.

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Niu H, Wu B, Peng Y, et al. Erratum to: RNA interference-mediated knockdown of RhoGDI2 induces the migration and invasion of human lung cancer A549 cells via activating the PI3K/Akt pathway. Tumour Biol. 2015;36(1):459doi: 10.1007/s13277-014-2904-y.
Niu, H., Wu, B., Peng, Y., Jiang, H., Zhang, Y., Wang, J., Zhang, Y., & He, P. (2015). Erratum to: RNA interference-mediated knockdown of RhoGDI2 induces the migration and invasion of human lung cancer A549 cells via activating the PI3K/Akt pathway. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, 36(1), 459. https://doi.org/10.1007/s13277-014-2904-y
Niu, Huiyan, et al. "Erratum to: RNA interference-mediated knockdown of RhoGDI2 induces the migration and invasion of human lung cancer A549 cells via activating the PI3K/Akt pathway." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. 36,1 (2015): 459. doi: https://doi.org/10.1007/s13277-014-2904-y
Niu H, Wu B, Peng Y, Jiang H, Zhang Y, Wang J, Zhang Y, He P. Erratum to: RNA interference-mediated knockdown of RhoGDI2 induces the migration and invasion of human lung cancer A549 cells via activating the PI3K/Akt pathway. Tumour Biol. 2015 Jan;36(1):459. doi: 10.1007/s13277-014-2904-y. PMID: 25487616.
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Letter of retraction.

Clinical and investigative medicine. Medecine clinique et experimentale

[No authors listed]
PMID: 26026640
Clin Invest Med. 2015 May 31;38(3):E142.

It has come to our attention that the a manuscript published in CIM: JianXin J, Cha Y, ZhiPeng L, Jie X, Hao Z, Meiyuan C, ChengYi S "GOLP3 is a predictor of survival in patients with hepatocellular carcinoma" Clin...

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Letter of retraction. Clin Invest Med. 2015;38(3):E142
(2015). Letter of retraction. Clinical and investigative medicine. Medecine clinique et experimentale, 38(3), E142.
"Letter of retraction." Clinical and investigative medicine. Medecine clinique et experimentale vol. 38,3 (2015): E142.
Letter of retraction. Clin Invest Med. 2015 May 31;38(3):E142. PMID: 26026640.
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MYSM-1 suppresses migration and invasion in renal carcinoma through inhibiting epithelial-mesenchymal transition.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Zhou L, Shi L, Guo H, Yao X.
PMID: 26409454
Tumour Biol. 2015 Sep 27; doi: 10.1007/s13277-015-4138-z. Epub 2015 Sep 27.

Renal cell carcinoma (RCC) is the most common malignant renal tumor and is prone to metastasis. However, the molecular variation and mechanism underlying renal cell carcinoma metastasis remains largely unknown. In our previous study, it was found that MYSM-1...

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Zhou L, Shi L, Guo H, et al. MYSM-1 suppresses migration and invasion in renal carcinoma through inhibiting epithelial-mesenchymal transition. Tumour Biol. 2015;doi: 10.1007/s13277-015-4138-z.
Zhou, L., Shi, L., Guo, H., & Yao, X. (2015). MYSM-1 suppresses migration and invasion in renal carcinoma through inhibiting epithelial-mesenchymal transition. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, . https://doi.org/10.1007/s13277-015-4138-z
Zhou, Lei, et al. "MYSM-1 suppresses migration and invasion in renal carcinoma through inhibiting epithelial-mesenchymal transition." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. (2015). doi: https://doi.org/10.1007/s13277-015-4138-z
Zhou L, Shi L, Guo H, Yao X. MYSM-1 suppresses migration and invasion in renal carcinoma through inhibiting epithelial-mesenchymal transition. Tumour Biol. 2015 Sep 27; doi: 10.1007/s13277-015-4138-z. Epub 2015 Sep 27. PMID: 26409454.
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Cancer/testis antigens as molecular drug targets using network pharmacology.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Kumar A, Sharma D, Aggarwal ML, Chacko KM, Bhatt TK.
PMID: 27709548
Tumour Biol. 2016 Oct 05; doi: 10.1007/s13277-016-5333-2. Epub 2016 Oct 05.

Present chemotherapeutic drugs have limited efficacy and severe side effects. Considering the complexity of cancer, an effective strategy is necessary to discover multiple new drug targets. Cancer/testis antigens are vital for cancer cell progression. We have performed a computational...

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Kumar A, Sharma D, Aggarwal ML, et al. Cancer/testis antigens as molecular drug targets using network pharmacology. Tumour Biol. 2016;doi: 10.1007/s13277-016-5333-2.
Kumar, A., Sharma, D., Aggarwal, M. L., Chacko, K. M., & Bhatt, T. K. (2016). Cancer/testis antigens as molecular drug targets using network pharmacology. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, . https://doi.org/10.1007/s13277-016-5333-2
Kumar, Anuj, et al. "Cancer/testis antigens as molecular drug targets using network pharmacology." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. (2016). doi: https://doi.org/10.1007/s13277-016-5333-2
Kumar A, Sharma D, Aggarwal ML, Chacko KM, Bhatt TK. Cancer/testis antigens as molecular drug targets using network pharmacology. Tumour Biol. 2016 Oct 05; doi: 10.1007/s13277-016-5333-2. Epub 2016 Oct 05. PMID: 27709548.
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Regulation of exosomes released from normal ovarian epithelial cells and ovarian cancer cells.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Zhang W, Yang J, Cao D, You Y, Shen K, Peng P.
PMID: 27714673
Tumour Biol. 2016 Oct 06; doi: 10.1007/s13277-016-5394-2. Epub 2016 Oct 06.

As important cell to cell communicator, exosomes carry a range of bioactive molecules which can significantly influence phenotype of recipient cells. Inhibiting or removing cancer cell-derived exosomes are of therapeutic interest. However, regulation of secretion and release mechanism of...

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Zhang W, Yang J, Cao D, et al. Regulation of exosomes released from normal ovarian epithelial cells and ovarian cancer cells. Tumour Biol. 2016;doi: 10.1007/s13277-016-5394-2.
Zhang, W., Yang, J., Cao, D., You, Y., Shen, K., & Peng, P. (2016). Regulation of exosomes released from normal ovarian epithelial cells and ovarian cancer cells. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, . https://doi.org/10.1007/s13277-016-5394-2
Zhang, Wei, et al. "Regulation of exosomes released from normal ovarian epithelial cells and ovarian cancer cells." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. (2016). doi: https://doi.org/10.1007/s13277-016-5394-2
Zhang W, Yang J, Cao D, You Y, Shen K, Peng P. Regulation of exosomes released from normal ovarian epithelial cells and ovarian cancer cells. Tumour Biol. 2016 Oct 06; doi: 10.1007/s13277-016-5394-2. Epub 2016 Oct 06. PMID: 27714673.
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Serum levels of apolipoprotein E correlates with disease progression and poor prognosis in breast cancer.

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine

Xu X, Wan J, Yuan L, Ba J, Feng P, Long W, Huang H, Liu P, Cai Y, Liu M, Luo J, Li L.
PMID: 27709551
Tumour Biol. 2016 Oct 05; doi: 10.1007/s13277-016-5453-8. Epub 2016 Oct 05.

ApoE has been reported to be associated with tumorigenesis and tumor progression. In this study, we explored the potential diagnostic and prognostic role of serum ApoE in breast cancer patients. Subject cohorts consisted of 152 normal healthy controls female...

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Xu X, Wan J, Yuan L, et al. Serum levels of apolipoprotein E correlates with disease progression and poor prognosis in breast cancer. Tumour Biol. 2016;doi: 10.1007/s13277-016-5453-8.
Xu, X., Wan, J., Yuan, L., Ba, J., Feng, P., Long, W., Huang, H., Liu, P., Cai, Y., Liu, M., Luo, J., & Li, L. (2016). Serum levels of apolipoprotein E correlates with disease progression and poor prognosis in breast cancer. Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, . https://doi.org/10.1007/s13277-016-5453-8
Xu, Xiangdong, et al. "Serum levels of apolipoprotein E correlates with disease progression and poor prognosis in breast cancer." Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine vol. (2016). doi: https://doi.org/10.1007/s13277-016-5453-8
Xu X, Wan J, Yuan L, Ba J, Feng P, Long W, Huang H, Liu P, Cai Y, Liu M, Luo J, Li L. Serum levels of apolipoprotein E correlates with disease progression and poor prognosis in breast cancer. Tumour Biol. 2016 Oct 05; doi: 10.1007/s13277-016-5453-8. Epub 2016 Oct 05. PMID: 27709551.
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Showing 1 to 12 of 244 entries